The 'clue' offered by the Nature Reviews folks, takes up PART of the way,
with epilepsy. What's missing is information on how to MODERATE the
inflammation.
My research shows that high-pH therapy is about THE BEST WAY to REDUCE the
suspected inflammation.
ANY interested individuals are invited to message me, for more information.
Thanks, and God Bless,
Dr. Dan, PhD Researcher
On Sat, Apr 23, 2011 at 10:09 PM, <Angls4Hope@aol.com> wrote:
>
>
>
> The role of inflammation in epilepsy
>
> Review
>
> Nature Reviews Neurology 7, 31-40 (January 2011) |
> doi:10.1038/nrneurol.2010.178
> Subject Categories: _Epilepsy_
> (http://www.nature.com/nrneurol/archive/neuro_s6_current_archive.html) |
> _Neuroimmunology and neuroinflammation_
> (http://www.nature.com/nrneurol/archive/neuro_s19_current_archive.html) |
> _Injury, repair and rehabilitation_
> (http://www.nature.com/nrneurol/archive/neuro_s13_current_archive.html)
> The role of inflammation in epilepsy
> Annamaria Vezzani, Jacqueline French, Tamas Bartfai & Tallie Z. Baram
> _About the authors_
> (
> http://www.nature.com/nrneurol/journal/v7/n1/authors/nrneurol.2010.178.html)
>
>
>
> (
> http://www.nature.com/nrneurol/journal/v7/n1/abs/nrneurol.2010.178.html?lang=en#top)
> Abstract
> Epilepsy is the third most common chronic brain disorder, and is
> characterized by an enduring predisposition to generate seizures. Despite
> progress
> in pharmacological and surgical treatments of epilepsy, relatively little
> is
> known about the processes leading to the generation of individual
> seizures, and about the mechanisms whereby a healthy brain is rendered
> epileptic.
> These gaps in our knowledge hamper the development of better preventive
> treatments and cures for the ≈30% of epilepsy cases that prove resistant to
>
> current therapies. Here, we focus on the rapidly growing body of evidence
> that
> supports the involvement of inflammatory mediators—released by brain cells
> and peripheral immune cells—in both the origin of individual seizures and
> the epileptogenic process. We first describe aspects of brain inflammation
> and immunity, before exploring the evidence from clinical and experimental
> studies for a relationship between inflammation and epilepsy. Subsequently,
>
> we discuss how seizures cause inflammation, and whether such inflammation,
> in turn, influences the occurrence and severity of seizures, and
> seizure-related neuronal death. Further insight into the complex role of
> inflammation in the generation and exacerbation of epilepsy should yield
> new molecular
> targets for the design of antiepileptic drugs, which might not only
> inhibit the symptoms of this disorder, but also prevent or abrogate disease
>
> pathogenesis.
>
>
> (
> http://www.nature.com/nrneurol/journal/v7/n1/abs/nrneurol.2010.178.html?lang=en#top)
>
>
> Author affiliations
> A. Vezzani, J. French, T. Bartfai & T. Z. Baram
> Department of Neuroscience, Mario Negri Institute for Pharmacological
> Research, Via Giuseppe La Masa 19, 20156 Milan, Italy (A. Vezzani). NYU
> Comprehensive Epilepsy Center, 223 East 34th Street, New York, NY 10016,
> USA (J.
> French). Molecular and Integrative Neurosciences Department, The Scripps
> Research Institute, 10550 North Torrey Pines Road, La Jolla, CA 92037, USA
> (T. Bartfai). Pediatrics, Anatomy and Neurobiology, and Neurology, UC
> Irvine,
> ZOT 4475, Irvine, CA 4475, USA (T. Z. Baram).
> Correspondence to: A. Vezzani _vezzani@marionegri.it_
> (mailto:vezzani@marionegri.it)
> Published online 7 December 2010
>
> [Non-text portions of this message have been removed]
>
>
>
[Non-text portions of this message have been removed]
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