Body Fitness Health

Wednesday, September 8, 2010

[AlternativeAnswers] IV Vitamin C and Swine Flu success stories....

IV Vitamin C and Swine Flu success stories....

IV Vitamin C is now proven effective against Swine Flu but why not also
have the best tolerated oral form of vitamin C available anywhere on hand,
BioEn'R-G'y, in your patients homes along with the most powerful colloidal
silver available anywhere, ACS 200. Nothing compares to the proven
killing power of ACS 200.

Let's keep both products in the home of your patients and let them know
you are available to also give IV Vitamin C that is proven to work. But
since sometimes people get ill when there is no way to get an IV, let's all be
prepared.

Dr Abe Ber MD (H.) also has a well documented case of Swine flu in a very
sick young girl recently. He treated her just once with 50 GM of IV
Vitamin C. Three days later she was back in school!

We all know that this Swine Flu story is being used to frighten people.
Yet compared to normal death rates from regular flu, all the fuss makes no
sense. It appears now to primarily be just a scheme to foist a useless and
clearly dangerous $1 billion worth of untested Swine Flu vaccine on the
public, as it would be a shame to just throw it away and poison some area of the
earth with all the mercury and other toxins in it.

By pretending that flu vaccines work better than Vitamin C, and in the
name of possible saving a couple of lives, the authorities are willing to
chance doing irreparable harm to thousands of us. This is done with no real
warning to all of those unsuspecting and trusting people. The NBC special on
Andrew Wakefield research on vaccine danger was another whitewash that
refused to tell the truth about his documentation about the autism connection to
vaccines.

Vitamin C in high enough doses will provide all the protection needed by
99% of the population without any of the risks associated with the use of
vaccines. But since not everyone can get an IV Vitamin C when they need it we
need to help our patients to be prepared at home with ACS 200 and a couple
bottles of BIOEN'R-G'Y C.

Fortunately BIOEN'R-G'Y C is so well tolerated that most can take 20
grams a day by mouth with minimal increase in stools and probable high levels
in urine testing ( see Bright spot) so that clearly they are going to
recover in far less time than anyone not getting aggressive well tolerated form
of oral vitamin C.

We now have another slight piece of incorrect information do deal with.
Dr Mercola, my friend, has warned the public against using colloidal silver!
He says it might work but he feels it is a heavy metal so you might have
to take some heavy metal treatment in your life. But with the zeolite we
all need to deal with our mercury and lead exposures this is not a serious
situation. In fact, the evidence is persuasive that silver is a conditionally
essential nutrient and not a toxic heavy metal at all.

But unfortunately he then assumes that the normal healing or cleansing
Herxheimer reactions that some patients have reported when they have a massive
die off of their infections, is or might be a a "cytokine storm" that
could hurt the lungs and might even kill you. Yes, everything we do has a
benefit and a risk but here the proven benefits of ACS handling every
infection it has ever been tested against clearly would be a very strong
benefit against a risk that has never been reported anywhere in the world in the
humans and animals receiving it for many years now.

I feel that this statement is just an unsupportable supposition that I
know when Dr Mercola realizes how many can die without an effective
alternative like BIOEN'R-G'Y C or ACS 200, he will correct his position. 100,000 die
each year of antibiotic resistant infections and silver and vitamin C
could save most.

ACS silver successfully kills all viruses and lab reports found on
www.resultsrna.com prove that ACS 200 is 240 times more effective against Staph
Aureus than ASAP, one of the most heavily promoted silver products on the
market and 20 times more effective against Candida than ASAP.

Dr Tom LEVY MD, expert in the treatment of infections with Vitamin C has
provided additional valuable information here about the use of IV Vitamin C
in swine flu.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com
To FACT and Dr. Gordon:

Hello,

I would like to submit a case report of a nearly terminal case of swine
flu completely cured in short order by vitamin C. The doctors in the FACT
group are very aware individuals, and they collectively have many brilliant
and innovative ways to approach treating their patients. However, I want to
make it clear that adequately dosed vitamin C, to my knowledge, has never
failed to cure an acute viral syndrome. Specifically, all these doctors
should now realized that H1N1, the swine flu virus, while perhaps proving to be
more potent than a host of other flu viruses, need not be a feared bogeyman
with vitamin C in their arsenal.

While I intend to assemble a more substantial case report from the
hospital chart in the future, here are the words of my colleague in New Zealand,
John Appleton:

"The short story is:

Waikato farmer goes to Fiji for holiday
Starts developing flu like symptoms--decides to tough it out
Arrives back in NZ very sick--swine flu
Tauranga Hospital not able to treat him (what was not known at the time is
that he has leukemia--he didn't know either)
Sent him to Auckland Hospital--continues to
deteriorate--Tamiflu--antibiotics etc. (usual stuff)
Brother-in-law (knows a bit about vitamin C) contacts Thomas Levy in the
US who refers him to me
I provided a lot of info on vitamin C etc and referred family to CAM
(Centre for Advanced Medicine) www.camltd.co.nz in Auckland
Family pushes to get him some IVC--hospital refuses
CAM doctors encourages hospital then to try vitamin C
Patient deteriorates further and is on life support--family told nothing
more can be done and life support will be switched off on Monday. Lungs not
functioning.
Family says NO--until everything has been tried--they won't agree to life
support being 'switched off'.
Hospital is pushed hard to give him IVC and reluctantly they agree. (50
grams twice a day I think) saying if no improvement by Friday that's it
Patient shows signs of improvement by Wednesday--hospital very surprised
Concerns expressed about kidneys (which we anticipated)
New specialist wants to stop vitamin C--family is told liver is failing
'caused by vitamin C'. I give them lots of data to say liver is more likely
to be affected by antibiotics.
Patient recovery continues to the point where he can be transferred to
Waikato (closer to home); on ventilator and NG tube feeding
Doctors there more receptive to vitamin C but won't agree to continue as
per Auckland
Family gets (name deleted) high profile lawyer involved who writes letter
about patient rights and rings hospital to recommend that they can either
sort it out with the family or......?
Hospital continues with VC albeit at a much lower dose. CAM doctor travels
to Waikato to endorse and recommend IVC at higher doses. I have heard that
the lawyer was shocked at what she learned about hospital system
Patient continues to recover--now conscious (thinks he has only been in
hospital 3 days)--now 8 weeks in total
Hospital staff stunned--never seen anything like this
Patient is told by brother-in-law that VC has saved him
Family absolutely blown away at what has gone on. Wife has not lost her
husband and children have their father.
Patient now fully 'with it' and is talking normally with family and taking
Lypospheric vitamin C (6 grams daily)"

For those doctors wondering about doses, I communicated directly with the
brother-in-law. He informed me that on the Tuesday following the initial
"deadline" 25 grams was given intravenously. On Wednesday, 25 gram infusions
were again repeated twice. Thursday the patient received 75 grams, and
starting on Friday he received 100 grams intravenously and stayed at this dose
daily for another 4 to 6 days. Then the new consultant had the vitamin C
discontinued completely. One week later, the IVC was restarted at only one
gram twice daily.

Please resend this case history to any and all who you think could
benefit, including your friends and contacts in our government. The latest info on
the swine flu indicates it certainly has the potential capacity to become
a great killer. This does not have to be the case. Obviously, a reasonable
daily dose of vitamin C could be expected to do an even better job at
preventing H1N1 while having no downside relative to the mass vaccinations
getting ready to take place.

Below find the link to the New Zealand 60 Minutes show on the "terminal"
advanced swine flu patient cured with intravenous vitamin C. Also note
toward the end of the clip that the patient's hairy cell leukemia "disappeared"
as well. The abilities of properly utilized vitamin C are slowly but surely
beginning to be recognized.

http://www.3news.co.nz/Living-Proof/tabid/371/articleID/171328/Default.aspx

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[AlternativeAnswers] Clinical presentation of childhood neuroborreliosis; neurological examination ma

_Arch Dis Child._
(javascript:AL_get(this,%20'jour',%20'Arch%20Dis%20Child.');) 2010 Jun 28. [Epub ahead of print]
Clinical presentation of childhood neuroborreliosis; neurological
examination may be normal.
_Broekhuijsen-van Henten DM_
(http://www.ncbi.nlm.nih.gov/pubmed?term="Broekhuijsen-van%20Henten%20DM"[Author]) , _Braun KP_
(http://www.ncbi.nlm.nih.gov/pubmed?term="Braun%20KP"[Author]) , _Wolfs TF_
(http://www.ncbi.nlm.nih.gov/pubmed?term="Wolfs%20TF"[Author]) .
Department of Paediatrics, Isala Clinics, Zwolle, The Netherlands.

Abstract
Objective
Neuroborreliosis has its highest incidence in children and the older
people. Signs and symptoms are different between the different age groups. The
aim of this study was to describe the clinical spectrum of neuroborreliosis
in children.
Design
The Dutch Paediatric Surveillance system registered cases of childhood
neuroborreliosis during 2 years. All Dutch paediatric hospitals took part in
this surveillance. Criteria for reporting cases were strictly defined.
Results
89 cases of neuroborreliosis were reported; in 66 cases diagnosis was
confirmed. Facial weakness was one of the presenting symptoms in 47 cases (71%)
and the only symptom in nine children (14%). The five complaints most
frequently reported were: malaise, headache, fatigue, fever and neck pain. 52
children (79%) had one or more objective neurological signs at presentation,
of which facial nerve palsy, other cranial nerve abnormalities and
meningeal signs were most frequent. 14 patients (21%), however, had no
neurological signs at physical examination. In these patients, the number of
subjective complaints was higher, and the time interval to diagnosis was longer
compared with those with objective neurological abnormalities.
Conclusions
In this study, 79% of paediatric neuroborreliosis patients presented with
neurological signs, most often facial nerve palsy. 21% presented in an
atypical way without neurological signs. A thorough neurological examination is
essential once neuroborreliosis is considered in children. Even in the
absence of neurological signs, neuroborreliosis may be suspected in children
with typical antecedents and multiple symptoms. Cerebrospinal fluid
investigations are then required to confirm the diagnosis.
PMID: 20584849 [PubMed - as supplied by publisher]

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[AlternativeAnswers] Insanity virus

The June issue of Discover magazine has an article on the insanity virus
and human endogenous retrovirus and bipolar, MS and Schizophrenia. Now we
have more evidence supporting the infection component of my FIGHT program.

This research about the "insanity" virus has not stopped, and in fact, the
more you read up on endogenous retrovirus the more you will see that this
all further reinforces my FIGHT4yourhealth concept. The June Discover
magazine on the newsstands brings this story up to date but the more you become
interested in the infection component of today's epidemic of impaired
health, the more you will see how this infection from endogenous retroviruses
found in what we used to call our JUNK DNA, helps explain Bipolar and MS as
well as Schizophrenia.

Maybe we all need to get toxins out so our immune system can handle these
inborn infections better, and more of us will need to lower the total body
burden of all infections fungal bacterial and viral using ACS 200 Silver
that is proven to efficiently lower even Borrelia and Candida.

This is worth really understanding, as this is real and when we begin to
understand how these virus that are in our DNA are kept under control, until
certain things happen, like a severe viral infection during pregnancy, and
then years later the child starts to hear voices
etc. I have covered on my website with 8 hours of webinars, the topics of
Food, Infection, Genetics, Hormones, Toxins, etc but this aspect of
infection was not covered. If we think about it, there could be some tie in here
to the live virus given children when they receive their MMR, and subsequent
development of Autism. There could be a HERV-W involved in that condition
too.

Garry F. Gordon MD,DO,MD(H)
President, Gordon Research Institute
www.gordonresearch.com

#1: http://discover.coverleaf.com/discovermagazine/201006/?pg=64#pg65

#2:
http://articles.sfgate.com/2002-08-05/news/17557040_1_mental-illness-mental-health-dr-e-fuller-torrey

Insanity virus -- a crazy idea? / Mainstream psychiatric outcast ponders
parasitic mental illness
August 05, 2002|By Keay Davidson, Chronicle Science Writer

#3: New Findings Boost Theory That Infection Causes Schizophrenia

Psychiatric News March 19, 2010
Volume 45 Number 6 Page 1
© American Psychiatric Association
1. Mark Moran

A review of studies of maternal exposure to infectious agents and
schizophrenia in their offspring suggests that eliminating certain infections could
prevent as many as 30 percent of schizophrenia cases.

Call it an instance of science being stranger than science fiction, and of
nature's unintended consequences.

In 2000, a team of British researchers published a remarkable paper in the
journal of the Royal Society titled "Fatal Attraction in Rats Affected
With Toxoplasma Gondii." It seems that rodents infected with Toxoplasmosis
gondi (T. gondii), a parasite that normally thrives in cats, become fatally
attracted to cat urine, causing them to shed their normal avoidant behavior
in the presence of a cat.

Naturally, cat catches rodent and devours it, with the result that the
parasite T. gondii is again where it belongs: in a feline host.

Observing this case of attraction gone fatally wrong, the researchers,
from the University of Oxford, postulated that T. gondii, one of nature's most
successful organisms, had developed an ingenious evolutionary mechanism
for manipulating the behavior of the rodent—in whom the parasite would have
reached a dead end—so that the rodent seeks out, suicidally, the feline host
in which the parasite can thrive and complete its life cycle.
Fast forward to 2009 when another group of researchers seeking to explore
the neurochemical mechanisms for the parasite's behavioral manipulation of
its host discover a likely culprit. In a paper published in the March 2009
PLoS One, Glenn McConkey, Ph.D., and colleagues at the University of Leeds
found that the genome of the T. gondii parasite encodes the enzyme tyrosine
hydroxylase, the central enzyme in dopamine synthesis.
"Intriguingly, dopamine is the most logical neurotransmitter candidate for
altering the avoidant behavior of the rodent," McConkey told Psychiatric
News. "So it would make sense for the parasite to increase dopamine. For the
parasite, this is an ideal way to manipulate the behavior of the host."

New Credibility for Infectious Etiology
And this strange cat-and-mouse story has yet another wrinkle.
For decades, an excess of dopamine has been implicated in schizophrenia.
And so last year's discovery that T. gondii synthesizes dopamine has lent
new credence to an intriguing, though not universally accepted, theory—that
at least some forms of schizophrenia may be caused by infection.

Among researchers inclined to the theory, T. gondii has been one, among
several, of the most prominent candidates.

"Humans infected with T. gondii will form the same cysts on the brain as
are found in infected rodents when high levels of the gene involved in
dopamine synthesis are expressed," McConkey said. "While production of dopamine
in the rodents could be an evolutionary mechanism devised by the parasite,
in humans it would be merely accidental. But now suddenly, this provides a
possible link to the observed correlation of T. gondii with some forms of
schizophrenia."

The theory of an infectious etiology for at least some forms of
schizophrenia has a long history, though it is still outside the mainstream. As far
back as the 1960s, British epidemiologist Edward Hare, Ph.D., documented a
marked increase in late-winter and early-spring births of people with
schizophrenia, a finding that pointed to an infection, possibly acquired during
winter months in utero.

Psychiatrist E. Fuller Torrey, M.D., a long-time proponent of the theory,
published a paper in the Lancet as early as July 1973 titled "Slow and
Latent Viruses in Schizophrenia."
Since then, Torrey has published a number of papers with Johns Hopkins
University pediatrician and virologist Robert Yolken, M.D., showing increased
antibodies to T. gondii—possibly acquired in utero or early childhood
through exposure to cat feces, undercooked meat, or contaminated water, dirt, or
sand—among people with schizophrenia.
Meanwhile, infectious agents have been implicated in other diseases—the
heliobacter pylori virus in peptic ulcers, the papilloma virus in cervical
cancer—that had been considered unlikely candidates for infection. "It's no
longer a theory from Mars," Torrey told Psychiatric News. "We've arrived on
planet Earth."

Epidemiologic Data Accumulate
Now, a report published online in AJP in Advance on February 1 by Alan
Brown, M.D., of Columbia University and colleagues, reviews more than 40
studies looking at seroepidemiologic findings, ecologic data, and maternal
reports on prenatal exposure to influenza and other infectious agents and the
development of schizophrenia.

They found that odds ratios for associations between schizophrenia in
offspring and serologically documented maternal influenza, elevated levels of
antibody to T. gondii, and other peri-conceptional genital or reproductive
infections ranged between 3.0 and 5.0.
The study, "Prenatal Infection and Schizophrenia: A Review of
Epidemiologic and Translational Studies," was done in collaboration with Catherine
Schaefer, Ph.D., director of the Kaiser Permanente Research Program on Genes,
Environment, and Health, and Barbara Cohn, Ph.D., director of the Child
Health and Development Studies, funded primarily by the National Institutes of
Health.

In a study published in the April 2005 American Journal of Psychiatry,
titled "Maternal Exposure to Toxoplasmosis and Risk of Schizophrenia in Adult
Offspring," Brown and colleagues conducted serological assays for
Toxoplasma antibody on maternal serum specimens from pregnancies giving rise to 63
cases of schizophrenia and other schizophrenia spectrum disorders and
compared them with assays from 123 matched healthy subjects.

They found that the adjusted odds ratio of schizophrenia/schizophrenia
spectrum disorders for subjects with high maternal Toxoplasma IgG antibody
titers was 2.61.
"The infectious theory has garnered more interest in the research
community," Brown told Psychiatric News. "This is being driven in part by
accumulating data from epidemiological studies of schizophrenia and translational
neuroscience in which offspring exposed prenatally to immune challenge and
infection show evidence of neurobiological abnormalities found in
schizophrenia."

A Gene-Environment Interaction
Still, the pathogenic theory of schizophrenia runs counter to years of
orthodox thinking that schizophrenia is a congenital, organic brain disorder.
Maternal and intrauterine infections are notably common, so one question is
that if schizophrenia is infectious in origin, why aren't more offspring
born with schizophrenia?

But proponents acknowledge that infection alone is unlikely to cause
schizophrenia. "Infections are almost certainly interacting with susceptibility
genes and other environmental factors," Brown explained. "So
gene-environment interactions likely play a very important role."

Nor do proponents of the pathogenic theory insist that infection is
necessarily the cause of all schizophrenia.

"While replication in independent samples is warranted, the data from our
sample suggest that up to approximately 30 percent of schizophrenia cases
could be prevented in the offspring of the pregnant population [in the
review appearing in AJP in Advance] if we were to completely eliminate three of
the infections we studied—influenza, elevated Toxoplasma antibody, and
peri-conceptional genital-reproductive infections," Brown told Psychiatric
News.

While complete eradication is highly unlikely, Brown says, the finding has
implications for prevention and treatment (see Can Infection-Related
Schizophrenia Be Prevented?).
More "orthodox" researchers acknowledge that infection may be a factor in
a more expansive research vision looking at "domains of psychosis" in
which schizophrenia-like symptoms stem from different gene-environment
interactions.

"Most people acknowledge today that schizophrenia is never going to be
found to have a single etiology," said psychiatric researcher and AJP deputy
editor Carol Tamminga, M.D., of the University of Texas Southwestern Medical
Center. "It's more likely a syndrome like congestive heart failure than a
disease like Parkinson's with a single molecular lesion. So we can assume
that schizophrenia is a diagnosis with multiple etiologies and overlapping
risk factors.

"We know genetics and environmental factors are risks," she continued. "
Keeping an open mind, I think we will find a list of risk genes and a list
of environmental factors, and we will have to see how both of those play out
in producing symptoms of a brain disease like schizophrenia."

An abstract of "Fatal Attraction in Rats Infected With Toxoplasma Gondii"
is posted at <www.ncbi.nlm.nih.gov/pubmed/11007336>. "A Unique Dual
Activity Amino Acid Hydroxylase in Toxoplasma Gondii" is posted at
<www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0004801>. "Prenatal
Infection and Schizophrenia: A Review of Epidemiologic and Translational Studies"
is posted at
<http://ajp.psychiatryonline.org/cgi/reprint/appi.ajp.2009.09030361v1>. "Maternal Exposure to Toxoplasmosis and Risk of Schizophrenia in
Adult Offspring" is posted at
<http://ajp.psychiatryonline.org/cgi/content/full/162/4/767>.
http://pn.psychiatryonline.org/content/45/6/1.2.full

Related articles
Clinical & Research News: Can Infection-Related Schizophrenia be
Prevented? Psychiatr News March 19, 2010 45:19

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